Resistin as a predictive marker for myocardial injury in patients with acute myocardial infarction
Título del documento: Resistin as a predictive marker for myocardial injury in patients with acute myocardial infarction
Revista: Gaceta médica de Caracas
Base de datos:
Número de sistema: 000596114
ISSN: 2739-0012
Autores: 1
2
3
4
5
6
7
Instituciones: 1Department of Clinical Pathology, Faculty of Medicine, Hasanuddin University, Makassar 90245, Indonesia. Hasanuddin University Medical Research Center, Faculty of Public Health, Hasanuddin University, Makassar 90245, Indonesia.,
2Department of Clinical Pathology, Faculty of Medicine, Hasanuddin University, Makassar 90245, Indonesia.Dr. Tadjuddin Chalid General Hospital, Makassar 90241, Indonesia,
3Department of Clinical Pathology, Faculty of Medicine, Hasanuddin University, Makassar 90245, Indonesia. Dr. Wahidin Sudirohusodo General Teaching Hospital, Makassar 90245, Indonesia.,
4Dr. Wahidin Sudirohusodo General Teaching Hospital, Makassar 90245, Indonesia. Department of Cardiology and Vascular Medicine, Faculty of Medicine, Hasanuddin University, Makassar 90245, Indonesia. Hasanuddin University Teaching Hospital, Makassar 90245, Indonesia.,
5Department of Biostatistics, Faculty of Public Health, Hasanuddin University, Makassar 90245, Indonesia,
6Department of Cardiology and Vascular Medicine, Faculty of Medicine, Hasanuddin University, Makassar 90245, Indonesia,
7Department of Clinical Pathology, Faculty of Medicine, Hasanuddin University, Makassar 90245, Indonesia. Dr. Wahidin Sudirohusodo General Teaching Hospital, Makassar 90245, Indonesia. Hasanuddin University Teaching Hospital, Makassar 90245, Indonesia.,
Año:
Volumen: 132
Número: 3
Paginación: 719-727
País: Venezuela
Idioma: Español
Resumen en español High-sensitivity troponin I (hs-TnI) has recently been used as a reference marker to detect myocardial injury in acute myocardial infarction (AMI). Atherosclerotic plaque is the primary cause of AMI. Resistin plays a role in endothelial injury processes, inflammatory responses, lipid accumulation, progression of atherosclerotic plaque, plaque rupture, and cardiac remodeling after myocardial infarction, suggesting its potential relationship to the extent of myocardial injury. We aimed to assess whether resistin is comparable to hs-TnI in diagnosing acute myocardial injury.
Palabras clave: Resistin,
myocardial injury,
Hs-TnI,
acute myocardial infarction
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